Parkinson’s disease


Parkinson’s disease

Pathogenetic mechanisms of the development of Parkinson’s disease are explained by a lack of dopamine, a neurotransmitter which ensures the transfer of signals between neurons. A loss of dopamine-producing neurons occurs in the area of the brain known as “substantia nigra”. In the long term, we can observe a progressive gradual decrease of cognitive functions, change in the mental functions and an occurrence of psychotic symptoms.

Neurological symptoms of Parkinson’s disease include muscle rigidity, bradykinesis (reduced mobility), gait disorders and tremor at rest (shaking). Levodopa is administered during the traditional treatment, which is converted to dopamine in the brain, to compensate for its lack. In long-term administration, however, the effectiveness declines and it is necessary to increase the dose, which results in a more frequent occurrence of side effects.

Amino acid complexes have a different mechanism of action. They stimulate nerve cells to produce dopamine and prevent degenerative changes of neurons in the substantia nigra. This results in the reduction of neurological symptoms and improvement of rigidity, bradykinesis, gait and cognitive functions in patients with Parkinson’s disease. When administering amino acid compounds, administration of levodopa can be reduced, thus reducing the side effects of the treatment.

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Parkinson’s disease

Pathogenetic mechanisms of the development of Parkinson’s disease are explained by a lack of dopamine, a neurotransmitter which ensures the transfer of signals between neurons. A loss of dopamine-producing neurons occurs in the area of the brain known as “substantia nigra”. In the long term, we can observe a progressive gradual decrease of cognitive functions, change in the mental functions and an occurrence of psychotic symptoms.

Neurological symptoms of Parkinson’s disease include muscle rigidity, bradykinesis (reduced mobility), gait disorders and tremor at rest (shaking). Levodopa is administered during the traditional treatment, which is converted to dopamine in the brain, to compensate for its lack. In long-term administration, however, the effectiveness declines and it is necessary to increase the dose, which results in a more frequent occurrence of side effects.

Amino acid complexes have a different mechanism of action. They stimulate nerve cells to produce dopamine and prevent degenerative changes of neurons in the substantia nigra. This results in the reduction of neurological symptoms and improvement of rigidity, bradykinesis, gait and cognitive functions in patients with Parkinson’s disease. When administering amino acid compounds, administration of levodopa can be reduced, thus reducing the side effects of the treatment.

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